首页> 外文OA文献 >Quinolone resistance mechanisms in Salmonella enterica serovars Hadar, Kentucky, Virchow, Schwarzengrund and S. enterica spp. enterica serovar 4,5,12:i:- isolated from humans in Switzerland: Identification of a novel qnrD variant, qnrD2
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Quinolone resistance mechanisms in Salmonella enterica serovars Hadar, Kentucky, Virchow, Schwarzengrund and S. enterica spp. enterica serovar 4,5,12:i:- isolated from humans in Switzerland: Identification of a novel qnrD variant, qnrD2

机译:肠沙门氏菌血清中的喹诺酮耐药机制Hadar,肯塔基,Virchow,Schwarzengrund和S. enterica spp。从瑞士的人类分离得到的肠杆菌血清4,5,12:i:-:鉴定新型qnrD变体qnrD2

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摘要

Human isolates of Salmonella enterica serovars Hadar, Kentucky, Virchow, Schwarzengrund, and the monophasic variant of S. Typhimurium, Salmonella enterica subsp. enterica serovar 4,5,12:i:- were examined for mutations within the quinolone resistance target genes gyrA, gyrB, parC, and parE and for plasmid-mediated resistance genes. Differences were observed among the serovars. A novel variant of qnrD, qnrD2, was detected in an S. Hadar isolate.
机译:人类分离的肠炎沙门氏菌Hadar,肯塔基州,维尔茨豪,施瓦辛格隆德和鼠伤寒沙门氏菌的单相变种,肠炎沙门氏菌亚种。检查了肠血清4,5,12:i:-的喹诺酮抗性靶基因gyrA,gyrB,parC和parE中的突变以及质粒介导的抗性基因。在血清型之间观察到差异。在S. Hadar分离物中检测到qnrD的新型变体qnrD2。

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